Whole-exome sequencing reveals genetic variants in ERC1 and KCNG4 associated with complete hydatidiform mole in Chinese Han women

نویسندگان

  • Yan Yu
  • Bingjian Lu
  • Weiguo Lu
  • Shuang Li
  • Xiuqin Li
  • Xinyu Wang
  • Xiaoyun Wan
  • Yaxia Chen
  • Suwen Feng
  • Yao Jia
  • Ru Yang
  • Fangxu Tang
  • Xiong Li
  • Shulan Zhang
  • Xinyan Wang
  • Heng Wei
  • Zhilan Peng
  • Lin Lu
  • Huizhen Zhong
  • Linjun Zhao
  • Zhangqian Huang
  • Lin Lin
  • Weihong Shen
  • Yan Lu
  • Zhu Cao
  • Jian Zou
  • Yuejiang Ma
  • Xiaojing Chen
  • Qifang Tian
  • Shiming Lu
  • Pengyuan Liu
  • Ding Ma
  • Xing Xie
  • Xiaodong Cheng
چکیده

Complete hydatidiform mole (CHM) is a rare pregnancy-related disease with invasive potential. The genetics underlying the sporadic form of CHM have not been addressed previously, but maternal genetic variants may be involved in biparental CHM. We performed whole-exome sequencing of 51 patients with CHM and 47 healthy women to identify genetic variants associated with CHM. In addition, candidate variants were analyzed using single base extension and Matrix Assisted Laser Desorption/Ionization-Time of Flight Mass Spectrometry in 199 CHM patients and 400 healthy controls. We validated candidate variants using Sanger sequencing in 250 cases and 652 controls, including 205 new controls. Two single nucleotide polymorphisms, c.G48C(p.Q16H) inERC1 and c.G1114A(p.G372S) in KCNG4, were associated with an increased risk of CHM (p<0.05). These variants may contribute to the pathogenesis of CHM and could be used to screen pregnant women for this genetic abnormality.

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عنوان ژورنال:

دوره 8  شماره 

صفحات  -

تاریخ انتشار 2017